Major cardiac events were similarly present in both groups.
At time 0 after introduction of stretch injury, ligament laxity was present in both groups.
Comparison of these two lists identified 721 genes that were present in both groups (Fig. 5a), with 690 genes inversely regulated (Fig. 5b).
Exposure was determined by meconium assay and self-report with alcohol, marijuana, and tobacco present in both groups.
However, ligament laxity was still present in both groups two weeks after the injury and was not completely removed by growth in the immature group.
Reactive astrocytes were present in both groups, with the majority found at the lesion epicenter in the C1 group, whereas the C2 group demonstrated increased reactive astrocytes extending 1 mm caudal to the lesion epicenter.
Post-hoc tests indicated that the valence effect was present in both groups (sleep: t(45) = 30.4, P < 10−30; wake: t(24) = 16.0, P < 10−13), whereas subjective ratings did not differ systematically between the sleep and wake groups for either neutral (t 69) = 1.7, P = 0.09) or negative (t 69) = 0.8, P = 0.41) items, indicating no time of day effect on perceived valence.
The aim of this three-group study was to identify (i) whether FAR alterations are equally present in both groups, (ii) whether they are present rather during implicit or explicit FAR, (iii) and whether they are conveyed by similar or disorder-specific neural mechanisms.
Immunosuppression, a known risk factor for infection [4, 31 33], was equally present in both groups.
N2P2-habituation was present in both groups, but we found no significant N1-habituation.
Dorsolateral dysfunction was present in both groups of migraneurs, who also had higher rates of anxiety as compared to controls.
