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There were total 3 cell preparations treated with arsenite.
Gemma et al. (2003) observed reduced metabolism of low-concentration chloroform in human liver preparations treated with a CYP2E1 inhibitor.
The Figure 11 legend states that all preparations treated with dynamic clamp inhibition showed antidromic spikes but only a fraction that were not treated.
First, supernatants of PGN preparations treated with either native or heat-denatured LYS1 were used to trigger immune marker gene FRK1 expression in Arabidopsis seedlings.
In preparations treated with NR2d only we observed no recrossing fibers in three animals; this is consistent with the lack of detour observed electrophysiologically.
Importantly, none of the proteins were found to be upregulated by elevated preload but unaltered when preload was applied in the presence of CsA exhibited altered expression levels in unloaded preparations treated with CsA (data not shown).
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Here, both the PGN preparation treated with buffer only AND the PGN preparation treated overnight with LYS1 lack immunogenic activity in the respective supernatant.
Seven independent "sister" cultures (i.e. cultures derived from the same brain preparation), treated with EPO or control were analysed.
In contrast, a monomeric gp120 protein preparation treated with Peptide-N-Glycosidase F to reduce its glycan content, the algae protein PE, or HEL showed little specificity for these cells.
RNA preparations were treated with RNase-free DNase RQ1 (Promega Corporation, USA) and resuspended in DEPC-treated water.
The RNA preparations were treated with RNase-free DNase before their use in the reverse transcription.
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