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Yet, a recent study using spinal-cord slice preparation demonstrated that synaptic potentiation of the presynaptic afferents was mediated by NO released from glial cells, via mGluR1 activation [51].
Firstly, a study in the canine ventricular wedge preparation demonstrated that propranolol increased epicardial APD, yet shortened endocardial APD (Krishnan & Antzelevitch 1991).
Experiments performed using intact skin-nerve preparation demonstrated that proteolytic cleavage specifically ablates the mechanosensitivity of touch receptors and thus reproduces the effects observed in vitro.
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The resultant mixture achieved higher PCS glucan hydrolysis at lower enzyme loading than a culture filtrate from T. cellulolyticus or a commercial enzyme preparation, demonstrating that the five enzymes play a role as core enzymes in the hydrolysis of PCS glucan.
Studies with extensively purified preparations demonstrated that the agent contains a protein that is required for infectivity.
AFM analysis (Fig. S2) of Aβ preparations demonstrated that NaOH- and NH4OH-based protocols resulted in primarily oligomeric species with occasional fibrils and that HFIP-based protocols resulted in primarily oligomeric species with rare or absent fibrils.
Immunoblots and quantitative analyses of these preparations demonstrated that ozz−/− and heterozygous pups had a significantly higher proportion of MyHCemb in the insoluble myosin fraction than their corresponding wild-type littermates (Fig. 6C and D).
Specimens labeled with only one fluorochome and viewed with the instrument settings as used for double- or triple-labeled preparations demonstrated that there was no bleed-through between the detector channels under these recording conditions.
Our study demonstrated that mental preparation for RTW is not a linear process of improvement.
Another contraction to this model is that our study demonstrated that mental preparation is not a linear process of improvement.
Interestingly, after normalization for HIV-1 p24 content, no difference in infectivity was seen between these two viral preparations, demonstrating that the NA-mediated infectivity increase in influenza HA pseudoparticles is the consequence of NA activity on a substrate that is expressed at the surface of virions, rather that at the surface of target cells.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com