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In [14], the preferential partner selection in an evolutionary study of a prisoner's dilemma was studied.
CD4+ T cells polarisation into Th1, Th2, Th17, and Treg subsets is acquired depending on the tissue cytokine context and is revealed by the activation of specific transcription factors, the expression of several chemokine receptors, a specific cytokine expression profile for each subset, and the activation of preferential partner cells triggering specific immunity [ 56].
HER2 receptor, which is frequently overexpressed in breast cancer, is a preferential partner of EGFR and the formation of EGFR-HER2 heterodimers potentiates EGFR function by increasing EGF binding affinity, stabilizing and recycling the receptor on the membrane, and expanding the repertoire of intracellular signaling responses [ 95].
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In yeast, almost all B-type RRs tested (except RR19) present interactions with the three preferential partners, HPt2, 7, 9, of HK1, a putative poplar osmosensor.
Results showed that, depending on the environment, a legume could select its preferential rhizobial partner in order to establish symbiosis.
Its leading status as a preferential third partner country within FP7 is only contested by the USA.
The involvement of NRP1 is not surprising because this receptor is the preferential binding partner of Sema3A [25] [28].
This suggests that in cases where genes are under the regulation of an ETS-IRF composite element (EICE), Spi-B and not PU.1 is the preferential IRF partner.
We have also convincingly demonstrated that ErbB3 is the preferential heterodimerisation partner of EGFR in pancreatic cancer cells (Frolov et al, 2007).
Consistent with RSV and TNFα inducing functionally distinct NF-κB binding complexes, AP-1 was recently shown to be the preferential cooperative partner with NF-κB in RSV-induced IL-8 expression [ 46], whereas in TNFα-induced expression NF-κB cooperates preferentially with NF-IL-6 [ 18, 19, 22].
We also showed that E2-2 is the preferential binding partner of the pro-neuronal bHLH protein HASH-1 in SH-SY5Y neuroblastoma cells [ 42], and the EMSA results presented in this paper, showing that E2-2 is the main E-protein binding a μE5-E-box, corroborate this observation.
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