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Chronic heart failure (CHF) can be caused either by a predominant abnormality in systolic function (systolic heart failure) or a predominant abnormality in diastolic function (diastolic heart failure).
CHF caused by a predominant abnormality in diastolic function has been increasingly recognised as a common entity and a cause of significant morbidity and mortality, as 40 50% of the patients with heart failure may have isolated diastolic dysfunction with a normal or near normal left ventricle (LV) systolic function [2, 3, 4].
This suggests that alteration of hepatic lipid storage represents an early and predominant abnormality in this cohort.
Analysis of sperm morphology revealed that MeHg group had lower percentage of morphologically normal spermatozoa, whereas the predominant abnormality was in the sperm head.
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The predominant abnormalities at weaning were fetlock valgus (19.1%), club feet (13.0%) and fetlock varus (11.2%).
Three predominant abnormalities were observed, including white matter signal abnormalities (19/69), major dilated Virchow Robin spaces (12/69) and temporal lobe abnormalities (20/69).
The predominant abnormalities were detrusor sphincter hyperreflexia and dyssynergia, findings consistent with HTLV-I-induced neurogenic bladder.
Although multiple immunologic abnormalities can contribute to the emergence and amplification of disease, the predominant abnormalities will probably vary in different patients.
The predominant abnormalities encountered in these studies were detrusor sphincter hyperreflexia (13/21 [61.9%]) and dyssynergia (4/21 [19%]); two patients (9.5%) were diagnosed with stress incontinence and two (9.5%) had completely normal urodynamic studies.
In CT scans of talc pneumoconiosis, the predominant abnormalities have been described as small centrilobular and subpleural nodules and conglomerated masses containing focal areas of high attenuation (Chong et al. 2006; Marchiori et al. 2004).
CMs are classified according to the predominant structural abnormality in skeletal muscle.
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