Effective sevoflurane preconditioning was defined as a significant effect of sevoflurane preconditioning on rSO2 measured by NIRS during IPC and on rSO2 measured by NIRS during postoperative reperfusion.
Ischemic or volatile anesthetic preconditioning is defined as tissue protection from impending ischemic cell damage by repetitive short periods of tissue exposure to ischemia or volatile anesthetics.
Effective ischemic preconditioning was defined as two cycles of a statistically and clinically significant decrease and increase of tissue oxygen saturation measured by NIRS.
The preconditioning algorithms are defined in the form of both a V-cycle and W-cycle and applied to solve challenging neutron diffusion problems.
Ischemic preconditioning (IPC) is defined as protection from ischemic cell damage by preceding repetitive short periods of ischemia followed by reperfusion [ 2].
The most powerful intervention to limit myocardial infarct size, other than early coronary reperfusion, is ischemic preconditioning, which is defined as a reduction in infarct size by a preceding short period of myocardial ischemia [ 7].
6 Ischemic preconditioning (IPC) is defined as brief episodes of ischemia followed by a long period of ischemia, representing the most powerful endogenous mechanism against the injurious effects of ischemia.
Ischemic preconditioning (IP) is defined as an experimental technique that renders tissues resistant to the deleterious effects of ischemia reperfusion by prior exposure to brief repetitive cycles of vascular occlusion [ 61].
Thus, preconditions for COC is defined as easy access to a range of needed services accompanied by high quality information and having the services that are needed to move forward.
The biological basis of preconditioning continues to be defined, and might involve signaling by Akt [ 113] and/or extracellular signal-related kinase 1/2 [ 114], and possibly an upregulation of PHD-2 during the hypoxic phase [ 115].
