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After 12 h exposure (time used in posterior experiments) viability was assessed using the crystal violet technique [ 18, 19].
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Model violations were measured by posterior predictive experiments, as implemented in PhyloBayes 3.0 [ 43].
Statistical measurements of fit and posterior predictive experiments enabled the adequacy of model assumptions to the data to be checked.
Repeated-measures ANOVAs revealed a significant interaction between seed regions (∆al-EC/pm-EC vs ∆PRC/PHC) × longitudinal subiculum section (anterior vs posterior) for Experiment 1 (F(1,14) = 7.3, p = 0.017) with a similar trend evident in the dataset of Experiment 2 (F(1,13) = 3.4, p = 0.087).
In the MACM analysis for the posterior cluster, 447 experiments (from 316 published studies) were included, with a total of 4279 participants.
The posterior distributions for Experiments 2A and B suggest that adding more distractors had a much smaller effect on (z RTs in the singleton search task (2A) than the conjunction search task (2B).
Since in our experiments posterior compartments were usually enlarged, we considered the possibility that non-autonomous apoptosis is the consequence of a size compensation mechanism across compartments to maintain the overall size of the disc.
Looking at Figure 4 and the scalp topographies, it can be seen that this interaction is mostly caused by a greater figure effect over the posterior region in Experiment 1. Outside this region, the modulation of the figure effect is generally similar across experiments.
In a third set of participants, we found a negative relationship between autistic traits and RS to simple geometric shapes in object-selective LO and posterior fusiform gyrus (Experiment 4).
In the original experiments, a posterior malleolar fracture or posterior talofibular ligament rupture was named stage three.
In this section, we only comment on experiments where posterior predictive tests were not rejected (i.e. there was significant violation of model assumptions).
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