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It might be possible that the reduction of chilled-induced fruit browning by GB could be associated to a protection of the membrane integrity and protection of photosystem II.
Since Bap31 interacts with a wide variety of proteins it was possible that the reduction of Bap31 surface levels was due to MARCH-VIII binding to Bap31 in intracellular membrane compartments.
It is possible that the reduction of 2Fe[24] is limited kinetically.
Alternatively, it is possible that the reduction of a single double bond alters the reducing potential of the MK.
It is possible that the reduction of atherosclerosis in clinical trials is at least partly due to the reduction of E-selectin levels.
It is certainly possible that the reduction of expression of FOXE1 in birds has resulted in the loss of these functional sequences.
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Another possible reason is that the reduction of tour, entertainment, and socializing with aging may directly cut down both trip frequency and length.
One possible explanation is that the reduction of lymphatic sprouting in Nrp2 mutants may reflect the lack of VEGFC/VEGFR3/NRP2-mediated positive signaling.
Rather, a first possible scenario is that the reduction of the choice and feeding scores for high sugar concentrations is caused by an inhibition from, for example, high-osmolarity sensors specifically upon the reflexive pathways.
It is therefore possible that the reduction in centromeric localization of Bub1 that we have observed in >75% of ESCO2-depleted cells is the cause of increased Sgo1 levels on chromosome arms in these cells.
It is also possible that the reduction in reported rates of infections may to some extent have been an artefact of reporting behaviours, since data were collected and reported by ICUs themselves and may have been influenced by perceptions of external scrutiny and performance management.
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