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To verify such possibilities, the effect of pH on the visible spectra of MB solution was tested in the absence of adsorbents.
To discriminate between these two alternative possibilities the effect of the chloride channel inhibitor DIDS, an efficient blocker of the VCC-formed channels [18], was evaluated on control and VCC-treated cells.
To distinguish between these two possibilities, the effect of mutations of the involved nucleotides on the cleavage rate and on the type of reaction (branching versus hydrolytic cleavage; Figure 4C) was assessed.
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To test this possibility, the effect of Vti1a and VAMP7 siRNAs on KChIP2-stimulated traffic of Kv4.2 was examined.
In order to explore this possibility, the effect of the RTK inhibitor genistein (25 μM) was tested.
Supporting this possibility, the effect of miR-17 on cell motility and invasion has been controversial in a cellular context.
To address this possibility, the effect of 10 μM K3[Fe CN 6] on the ability of etoposide quinone to enhance DNA cleavage mediated by the catalytic core was assessed.
In initial experiments to address this possibility, the effect of addition of cyclic GMP on the synthesis of cyclic di-GMP by purified XC_0249 was assessed.
To discard this possibility, the effect of the drug on the ATPase activities present in a microsomal fraction of MDCK cells was evaluated.
To investigate this possibility the effect of LMF on cyclic AMP production was determined in CHOK1 cells, which had been transfected with the human β3-AR.
To assess this possibility, the effect of bexarotene on tumour cell conditioned medium-mediated invasion of HUVE cells through Matrigel matrix was evaluated.
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