Exact(12)
In vitro cell culture showed that MC3T3-E1 cultivatedinathe in the pores of these scaffolds had positive proliferation potential.
No treatment was used as positive proliferation control.
A stimulation index ≥3 was considered as a positive proliferation response.
A lower dose of 0.25 μM As2O3, although tolerated, was unable to stimulate positive proliferation.
T-cell clones were generated from responder cells that gave a positive proliferation response.
The clonogenic assay in HEK293T cells identified positive proliferation and cellular survival after treatment with 0.5 μM As2O3 for 15 days.
Similar(48)
Usually, the percentage of PLK1/PLK3-positive cells was higher than the KI-67-positive proliferation fraction, so that there was no strict coexpression.
In addition to predictions for increased activity of positive cell proliferation mediators in data sets where cell proliferation was experimentally induced to increase, RCR also predicted decreased activities of negative regulators of proliferation.
In vitro cell culture indicated that MC3T3-E1 culturedltured on the CS-TSF/HAp composite had positive adhesion, proliferation, and differentiation potential.
Vessels were deemed positive for proliferation when they harboured at least one ki-67 positive cell and were observed inside the tumour mass or in its periphery.
NK cell education may include a combination of several types of mechanisms, from negative (cell-death based) through positive (cell proliferation-based) selection to fine-tuning via adjustment of the cell's ability to respond to activating stimuli.
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