"population alteration" is a correct and usable phrase in written English.
It refers to any change or modification in the size or composition of a particular population. Example: The government's policies have led to a significant population alteration in the city, with a decrease in the number of young families and an increase in elderly residents.
The research examines employment, fiscal position, elasticity, population alteration, and regional location as primary variables.
In the general population alteration in arterial function has been associated with the presence of vascular risk factors and is considered a marker of vascular disease associated with risk of future vascular events [47, 48].
In several mouse models and also from human population studies, alteration of p53 activity has been demonstrated to influence the comparison of premature/accelerate aging under some circumstances (such as stress) or otherwise induce tumor suppression [ 47- 52].
Carcinogens engender two of the major forces for evolutionary change: increased genetic diversity in a population and alterations of selective pressures.
The present study tested the hypothesis that changes in the resident endogenous cellular population accompany alterations in aortic collagen and elastin content during thoracic aortic aneurysm (TAA) development in a murine model.
Hence a novel mathematical framework is necessary to investigate the extent of heterogeneity of an asexually evolving population accumulating alterations with large fitness effects over time.
The development of BE has been thought to arise largely because of chronic esophageal inflammation and the subsequent the expansion of some sort of an altered stem cell population due to alteration of stromal niche factors.
In recent times, there has been an increase in interest surrounding it's use in various clinical populations demonstrating alterations in motor function.
In either case, the transfer of genetic information allows for prompt adaptation of cell populations to alterations in the microenvironment, and may help explain why hypoxia, irradiation, or cell activation augment microvesicle release [2].
Diabetes is a global health threat with an evolving disease morphology whereby onset, burden and course are shifting in concert with population-wide alterations in behavioral and lifestyle factors and disease management strategies [1] [2], [3].
Numerous interactions have been investigated including cross-talk between stromal and epithelial cell types, localized enrichment of endocrine and growth factor signaling within cell populations, and alterations cell-matrix associations observed in epithelial-mesenchymal transition.
