Sentence examples for poor reactivation from inspiring English sources

Indeed, BoHV-1 is able to establish latency in goats but with a poor reactivation success rate [ 30, 31].

Despite this, other studies have observed poor reactivation competence of LAT-negative virus when latent virus DNA loads are comparable to WT virus both in mice (Leib et al., 1989; Hoshino et al., 2009) and rabbits (O'Neil et al., 2004), suggesting that LATs influence latency during multiple phases of infection.

Strain McKrae is known for highly efficient reactivation following TCIE (the recovery of infectious virus in both the eye and TG occurs as early as 24 h post-treatment with 80 100% efficiency [31] [33]), while strain KOS is typically known as a poor reactivator (reactivation efficiency for KOS is 5 20% following TCIE [34], [35]).

Regarding preemptive treatment, immunological assessment of IFN-γ produced by CMV-specific CD8+ T cells and NK cell function as mentioned in virulence section might be a strong trigger to start preemptive treatment for detected CMV, because there is evidence that impaired CMV-specific CD8+ T cells IFN-γ production prior to CMV reactivation predicts poor outcome, which favors treatment.

And is there a quantitative association between CMV reactivation and poor outcome?

Is CMV reactivation associated with a poor outcome?

In particular, this study implicated limited telomerase function (possibly arising from poor activation by pluripotency factors) and reactivation of exogenous pluripotency factors in response to loss of full telomerase function, in addition to ALT in contributing to telomere length variability in iPSC reprogramming.

It was found that salicylaldoximes were able to quickly cleave the P S bond of organophosphate and avoid the reinhibition phenomenon in the reactivation process, but they lacked reactivating ability due to poor affinity for AChE.

Magnitude and persistence of immunological alterations over time have been associated with poor clinical outcomes: development of nosocomial infections, viral reactivation, and increased mortality [1].

Poor memory performance was found at both the time of memory reactivation (day 6 after training) and at testing of all groups that were designed as controls (day 7).

That's obviously a poor assumption because there was (a) attrition among actives and (b) reactivation of dormant accounts.