Sentence examples for polymorphism and the prevalence of from inspiring English sources

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The following studies suggest a link between PGC-1α and atherogenesis at the clinical level: Xie et al. reported a correlation between PGC-1α polymorphism and hypertension [14], and Zhang et al. showed an association between PGC-1α polymorphism and the prevalence of coronary artery disease [15].

However, in any genetic models, there were no statistically significant associations between each polymorphism and the prevalence of cerebral infarction.

The single association analysis revealed that there were no statistically significant associations between each polymorphism and the prevalence of cerebral infarction.

Phenotypic results of different co-inherited factor V Leiden heterozygous conditions in the presence of the novel heterozygous factor V Graz polymorphism and the prevalence of these conditions.

First, we performed the conventional single-association analysis, where the association between each polymorphism and the prevalence of cerebral infarction was examined individually.

However, there was no significant association between this polymorphism and the prevalence of cerebral infarction in any genetic models after Bonferroni's correction was applied (supplementary Table 1).

Similar(54)

Previous studies have revealed an association between interleukin 17A (IL17A) polymorphisms and the prevalence of rheumatoid arthritis (RA) in Japanese and Caucasian patients.

Similarly, regarding MMP9 C-1562T, F12 C46T, and VWF G-1051A polymorphisms, there were no statistically significant associations between these polymorphisms and the prevalence of cerebral infarction (supplementary Tables 2 4).

However, our pilot study included patients within a well-defined, specific population and was aimed to identify the presence of statistical associations between selected genetic polymorphisms and the prevalence of a specific disease.

Therefore, while HNF1A appears to influence CRP concentrations in the non-diabetic state, chronic elevation of CRP is unlikely mediating the association between the HNF1A polymorphism and the high prevalence of type 2 diabetes in this Aboriginal population.

Therefore, while HNF1A appears to influence CRP concentrations in the non-diabetic state, chronic elevation of CRP is unlikely mediating the association between the HNF1A polymorphism and the high prevalence of 2 diabetes in this Aboriginal population.

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