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The pervaporation results showed that AA plasma polymerization treated PUs had much higher selectivity for the separation of methanol from methyl-t-butyl ether (MTBE) than untreated and O2, N2 treated PUs.
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The effective phase diagrams observed in the simulations can be approximated by Flory—Huggins theory when the degree of polymerization is treated as an adjustable parameter.
To determine whether the loss of stable plasma membrane caveolae was a result of altered actin polymerization, we treated EGFP-caveolin-1-transfected cells with LatB and analyzed caveolar dynamics by spinning-disc and TIRF microscopy.
A literature example and an industrial polymerization reactor are treated to illustrate these properties.
For inhibition of actin polymerization, cells were treated with 0.2 μM LatB 30 min prior to live cell imaging.
To modulate actin polymerization, cells were treated with Latrunculin B (Calbiochem; 1 μM), Cytochalasin D (Applichem; 1 μM) for 24 h.
This result was associated with alteration in actin filaments polymerizations in the treated HT-29 cells.
To discriminate between de novo actin polymerization and reorganization of existing actin filaments, cells were treated with the actin polymerization inhibitor cytochalasin D (CytoD), prior to the incubation with PMA.
Subsequently, the GOD densely packed array on the PPF surface was subsequently treated by plasma polymerization (overcoating).
The pre-incubation of the B-cell line Dab-1 and the T-cell line Jurkat with non-toxic concentrations of these chemicals significantly decreased the phalloidin staining (Figure 2C) confirming that actin polymerization was prevented in the treated cells.
α-tomatine could also significantly alter polymerization of actin filaments in the treated PC-3 cells.
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