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Similar observations were seen for all spindle poisons (data not shown).
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CIV-hAGT cells could be successfully stained with MitoTracker DeepRed indicating the presence of a membrane potential, but mitochondria lost this dye after dissipation of the membrane potential by CCCP poisoning (data not shown).
Results appeared to be independent of pesticide poisoning, because we observed similar results when we excluded applicators who reported physician-diagnosed pesticide poisoning (data not shown).
Excluding applicators who reported physician-diagnosed pesticide poisoning did not change results (data not shown).
Indeed, washing TP53 -mutated BL41 cells exposed spindle poisons that allow them to regrow in a new medium (data not shown).
The incidence of death in the groups "Injury and poisoning" and "External causes of injury and poisoning" was 5.8% vs 3.5%and2.6%6% vs 1.5%, and remained significant even after adjusting for age (data not shown; P = 0.003 and P = 0.049, respectively).
In fact, some counties and cities don't report lead poisoning data to the CDC at all.
The lack of complete poisoning data from children aged 10 and 11 years meant we could not calculate an exact population incidence for the 10 – 14 years age group.
Acute poisoning data was extracted from patient charts.
The clinical researchers involved in the Morinaga dried milk poisoning did not collect any data on exposure levels or tissue concentrations.
Finally, in contrast to interfacial topoisomerase II poisons, covalent poisons did not enhance DNA cleavage mediated by the catalytic core.
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CEO of Professional Science Editing for Scientists @ prosciediting.com