Sentence examples for pneumonia pathogenesis from inspiring English sources

Studying children at all grades of severity would provide valuable insights into pneumonia pathogenesis.

Although HAP-associated bacteria may enter the lungs in several different ways, aspiration of bacteria from the mouth and oropharynx is perhaps the most important mechanism of lower-airway infection in pneumonia pathogenesis [ 10].

Animal and human studies have shown that preceding IAV infection enhances all aspects of S. pneumoniae pathogenesis from nasopharyngeal colonization to invasive pneumococcal disease7, leading to the strong predisposition to lethal secondary pneumococcal infection in IAV infected patients.

The significant role of the IPM in S. pneumoniae pathogenesis is indicated by the attenuated virulence of the mutant strain in intranasally infected mice [24].

However, the complexity of M. pneumoniae pathogenesis, which can include extrapulmonary spread, chronic sequelae such as asthma and COPD, and the potential for secondary infection, complicates diagnostic strategies.

Therefore, further studies are required to elucidate whether KpST66_1945 is implicated in K. pneumoniae pathogenesis.

One gene, pld1, was shown to be involved in virulence in a mouse model of pneumonia and revealed a novel implication of lipid metabolism in K. pneumoniae pathogenesis.

Strain Kp25.145 (a derivative of B5055, the reference strain of serotype K2) is a laboratory strain used to study K. pneumoniae pathogenesis [ 11, 21] and was chosen as the focus of this work.

Chlamydialike organisms in atherosclerotic lesions may explain controversies about the role of C. pneumoniae in pathogenesis of atherosclerosis (4 ).

Several reports have suggested a role of chronic C. pneumoniae infection in pathogenesis of CAD and other atherosclerotic syndromes [ 4, 5].

In this work, we have used a functional genomics-based approach to provide a comprehensive prediction of TF activation profiles and domain expression patterns of 3 different S. pneumoniae strains during pathogenesis.