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Fourth, resuscitation with consecutive inflammation might directly affect platelet reactivity.
Conclusion: Although interaction between platelets and the dialysis circuit increases platelet reactivity, continued dialysis decreases platelet reactivity.
Cardiogenic shock as well as reduced body temperature impacts platelet reactivity and its medical inhibition.
We evaluated plasma levels of cytokines, prostaglandins, and adenosine, and measured platelet reactivity during endotoxemia.
Platelet reactivity was reduced in ticagrelor and clopidogrel treated subjects, without any correlation with cytokine responses.
These patients are at high risk of persistent platelet reactivity under aspirin treatment.
The extent of luminal involvement of atherosclerotic vascular disease and platelet reactivity portend subsequent cardiovascular events.
Methods: Platelet reactivity was determined by flow cytometry in 36 patients with ESRD undergoing hemodialysis.
Several variants associated with PLT/MPV (PEAR1, MRVI1, PTGES3) were also associated with platelet reactivity.
Elderly patients display higher on clopidogrel platelet reactivity as compared with younger patients.
Hypertriglyceridemia increases platelet reactivity.
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