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Understanding the fitness consequences of climate-induced plasticity requires researchers to delineate plastic responses in experiments, create artificial phenotypes independent of genotype (Waddington 1953), and then track their long-term fitness in experimental or natural situations.
This phenomenon of 'pop-in' (displacement burst or strain burst) was attributed to very poor defect density prior to nanoindentation test so that the onset of plasticity requires load sufficient for dislocation nucleation and propagation.
In addition, this may be due, in part; pop-in events were attributed to the very poor defect density prior to the nanoindentation tests so that the onset of plasticity requires load sufficient for dislocation nucleation and propagation.
According to the needs of practical applications, balancing both strength and plasticity requires the following criteria for selecting an element for incorporation into an HEA system: matrix strength is improved by selecting an element with a VEC lower than the average VEC of the matrix, while ductility is improved by selecting another element with a VEC higher than the average VEC for the matrix.
The administration of ERK1/2 inhibitor PD98059 clearly demonstrates that cortical plasticity requires the activation of ERK1/2 signaling pathway.
Specificity of synaptic plasticity requires that cAMP activates PKA in a highly localized manner despite the speed with which cAMP diffuses.
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Tau is highly phosphorylated during development, reflecting the plasticity required during neuronal development and the requirement for a dynamic microtubule network (5, 19– 219.
Hebbian models of plasticity require temporal association between synaptic input and neuronal output to produce long-term potentiation of excitatory transmission.
Epigenetic marks, such as DNA methylation and histone modifications, play a fundamental role in the neuronal plasticity required for learning and memory.
It is well established that transcription factor combinations and the organization of cis-regulatory sequences control commitment to differentiation programs and preserve a nuclear plasticity required for neuronal functions.
The switch from hive work to foraging offers an opportunity for the integrative study of the physiological and genetic mechanisms that produce the behavioral plasticity required for major life history transitions.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com