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In patients with DM, VAT tertiles were not associated with vulnerable plaque (Table 6).
Unadjusted analyses revealed that children with presence of dental plaque, indicating poor oral hygiene, were more likely to present with enamel defects than their counterparts with less observable plaque (Table 4).
Values are presented as averages ± standard deviations for plaques collected 6 and 12 h after the last brushing In general, plaques collected after the use of CT and CPH had lower viabilities than the control plaque, while plaques collected after the use of ZC showed similar viability as the control plaque (Table 4).
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After adjusting for traditional cardiovascular risk factors adiponectin turned out to be significantly associated with calcified plaques (Table 6).
This was accompanied by a decrease in iNKT cell number in psoriatic plaques (Table 1).
Glial proteins, GFAP and excitatory amino acid transporter (EAAT) (Anderson and Swanson, 2000), were both detected in (human) senile plaques (Table 2); however, only EAAT was detected in TgCRND8 amyloid plaques (Table 1).
Moreover, these patients had lower log serum intact parathormone levels, LDL-cholesterol and albumin concentrations when compared with patients free of plaques (Table 1).
Brains selected as PA had no evidence of cognitive decline in the clinic, but had extensive cortical Aβ plaques (Table 1).
In this context it is interesting to note that the PE ether-linked lipids in the plaques were unique for Cx26, none being associated with Cx32 plaques (Table 3b).
DENV-1 and JEV but not DENV-2, -3, or -4 reactive antibodies were detected in post-infection sera from seroconverters by PRNT assay when titers were based on a 70% reduction in plaques (Table 1).
Using this strategy, four single gene deleted-mutant recombinants OV-IA82Δ002 [ 23], OV-IA82Δ024 [ 24], OV-IA82Δ120 (unpublished data), OV-IA82Δ121 [ 25] were finally generated and isolated through 17 and 20 rounds of plaque purification (each round takes at least 4 or 5 days to pick blue plaques, Table 1).
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