In mutant plants, defects were observed in ovules very early in development.
A large program has been performed in France in order to develop, for the design and operation of High Temperature Reactor plants, defect assessment and Leak-Before-Break (LBB) procedures.
Misexpression of miRNA target genes leads to plant developmental defects in meristems (HD-ZIPIII genes) [27], leaves (TCP) [28], flowers (AP2 genes; CORNGRASS1) [30], [30], and roots (NAC1) [31].
Single T-DNA insertion mutants, prf1-4 and prf2-1, showed very similar phenotypic effects, with plants showing defects in normal rosette leaf morphology as well as inflorescence development, leading to shorter overall plant height for these mutants (Fig. 1).
Instead, the Atcrt3 mutant plants displayed defects in elf18 responses, which were not observed to the same extent in the Atcrt1a crt1b double mutant plants (Figure 9, [27]; [27]).
Many higher plants use the day length as an environmental cue to switch from vegetative to reproductive growth and plants with defects in their circadian regulation cannot properly regulate the timing of the floral transition [ 46].
In addition, like rpl24b/stv1, eif3h mutant plants display defects in auxin responses.
The conditional RNAi approach to examine the function of AtCTF7 in plants revealed defects in sister chromatid cohesion in meiosis.
Overexpression of UGT73C6 led to the same phenotypic effects as observed in UGT73C5oe plants: growth defects indicative of BR deficiency.
These include plants with defects in syntaxin genes [ 43], and mutants with defects in the cytochrome P450 gene CYP83B1, which results in excess auxin synthesis [ 44].
