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This study was therefore projected to evaluate this plant acute toxicity effect on the hematological and biochemical parameters in order to validate its use.
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In ozone-sensitive plants acute exposure to this toxic pollutant leads to necrosis, caused by uncontrolled death of mesophyll cells [ 3, 4].
We previously showed that the aqueous extract of this plant possesses acute antihypertensive and vasorelaxant effects [ 14].
Five workers in an industrial waste plant developed acute toxic hepatitis, one of whom died after 3 months due to fulminant hepatitis.
The present study evaluated the effect of hydroponic pretreatment of strawberry (Fragaria x ananassa cv. 'Camarosa') roots with an H2S donor, sodium hydrosulfide (NaHS; 100 μM for 48 h), on the response of plants to acute heat shock treatment (42°C, 8 h).
In an attempt to understand Arabidopsis plant response to acute exposure to ether, twenty-five to thirty-day old Arabidopsis plants were expoldd to ether fumigArabidopsis4 h.
ETI is a more acute plant reaction often involving programmed cell death at infection sites.
VIGS with TRV NAG(N1) and TRV NAG(N2) constructs resulted in growth arrest and acute plant death.
Silencing of NbNAG by virus-induced gene silencing resulted in growth arrest and acute plant death with morphological markers of programmed cell death (PCD), which include chromatin fragmentation and modification of mitochondrial membrane potential.
Functional genomics using virus-induced gene silencing (VIGS) revealed that silencing of the N. benthamiana homolog of Neuroblastoma Amplified Gene (NAG), designated NbNAG, results in growth arrest and acute plant death.
To investigate endophyte effects on plant responses to acute water deficit stress, we did comprehensive profiling of plant metabolite levels in both shoot and root tissues of genetically identical clone pairs of tall fescue with endophyte (E+) and without endophyte (E-) in response to direct water deficit stress.
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