Multiphase flows consist of interacting phases that are dispersed randomly in space and in time.
Applied to each of several interrelated sub-systems, SIPOKS can, for example, usefully analyse an extended process into shorter phases and link the analysis of interacting processes from different hierarchical levels for a specific purpose [ 22].
In addition, during the S phase, Rif1 ensures that replication of interacting domains is temporally coordinated.
Nonequilibrium statistical physics, in particular the collective behavior of interacting particles near phase transitions, has already been recognized as very valuable for understanding counterintuitive evolutionary outcomes.
Here is the sequence of interacting events.
Phase synchronization implies the existence of a relationship between phases of twoweakly interacting (coupled) systems, whereas the amplitudes may remain uncorrelated.
For example, cyclin D controls the G1 phase by interacting with CDK4 and CDK6; cyclin E interacts with CDK2 and controls the end of the G1 phase and the transition through G1/S; the cyclin A/CDK2 complex regulates the S phase and the exit from it and the cyclin A/CDC2 CDK1) complex regulates the G2/M transition; cyclin B interacts with CDC2 and also regulates the G2/M transition.
The Tudor domain FMRP has been implicated in participating in DNA repair by specifically binding to H3K79me [ 6]. 53BP1 can control of S phase duration by interacting with the RB protein methylated at K810, maintaining its hypomethylated status [ 7], as well as it can bind to H4K20me2, a DSB mark [ 7].
FT-IR and XPS analyses show that organic and inorganic phases strongly interact mainly because of transesterification reactions involving the polyester chain and Ti atoms during hybrid synthesis.
A sharp interface Cartesian grid method for the large-eddy simulation of two-phase turbulent flows interacting with moving bodies is presented.
These data, together with results from Figures 4C and D, suggest that capsule formation may occlude or otherwise constrain the presentation of cell wall proteins and inhibit the interaction of plasminogen with surface receptors, although factors other than capsule formation may compromise the ability of stationary phase cells to interact with plasminogen.
