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In an attempt to decipher the factors which govern the phase of reactivation, some researchers adopted an immunossupressive model, in which reactivation of latent infection occurs following treatment with the immunossupressive reagent dexamethazone [ 68, 69].
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In combination with the interpretation of a UAV-based high-resolution digital surface model, multiple phases of reactivation are inferred.
Based on the seismic-stratigraphic architecture of the post-basement succession, we identify several phases of reactivation of the intrabasement structures associated with multiple tectonic events.
The structural setting of the hill mainly results from several phases of reactivation of the CI caldera faults which were active until about 9.5 ka.
The virus exists in a latent state, with periodic phases of reactivation, lytic replication and virus shedding.
Phase coupling of reactivations to a given theta oscillation implies a phase concentration of reactivation times.
Cells in this first phase of cell reactivation are considered as reactivating cells (RC).
In contrast, the K59 responsive (embryogenic) genotype was able to enter both the first and second phases of cell reactivation and was therefore able to undergo SE. β-Glc Y had little effect on the first phase of cell reactivation but proved to be a successful tool to reversibly block SE in the K59 genotype.
The first phase of cell reactivation involved several stages of cell reorganisation.
Further examination revealed that only the responsive genotype K59 was capable of undergoing the second phase of cell reactivation (Fig. 1D).
Observation of day 4 explants revealed that a small proportion of cells had started to undergo structural reorganisations associated with the first phase of cell reactivation (CR).
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