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During the oscillation phase metamorphosis and whisker transport were noticed.
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The transitional nature of phased metamorphosis differs from parallel and convergent evolution.
It affords a unique opportunity to study pathogen emergence via pathogroup conversion, and introduces phased metamorphosis as a new evolutionary concept.
The phylogeny and virulence potential of STEC O2 H6 compels us to propose a novel evolutionary concept whereby one pathogroup of E. coli undergoes phased metamorphosis into another.
The phylogeny of these E. coli and the repertoire of virulence traits they possess compel consideration of an alternate view of pathogen emergence, whereby one pathogroup of E. coli undergoes phased metamorphosis into another.
Furthermore, they have complex pelagobenthic life cycles which include development to a larval phase and metamorphosis to sedentary adult form [ 50].
In the second phase, after metamorphosis, heart development re-initiates and after final differentiation, the heart starts to beat and becomes functional.
Bursicon, another αβ heterodimer member of this cysteine knot family of neurohormones, was characterized in insects for its role in triggering the sclerotization of the cuticle and expansion of the wings during the final phase of metamorphosis [ 58].
Following a short nonfeeding pelagic phase, animals undergo metamorphosis, which is accompanied by body elongation and loss of the ciliated prototroch, telotroch, and neurotroch.
One possibility was that the loss of JH shortened a key growth phase and triggered premature metamorphosis, causing smaller larvae to transform into smaller adults.
In holometabolous insects, fully differentiated larval neurons exhibit maintenance growth during the larval stages and a second, post-embryonic phase of organizational growth during metamorphosis.
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