The phrase "persistent insult" is grammatically correct and can be used in written English.
It refers to a recurring or ongoing act of disrespect or offense towards someone. Example: Despite numerous warnings, the student continued to make persistent insults towards his teacher, resulting in his eventual suspension from school.
Thus, a common strategy to maintain sensory abilities against persistent environmental insult involves repair and regeneration.
In some cases, such patterns may be due to other adverse intrauterine conditions such as undernutrition 10 which may constitute a slight but persistent brain insult.
Thus, neuromasts are endowed with invariant and enduring regenerative capacity, which may have evolved for fish to maintain life-long sensory ability despite persistent environmental insult (Ciba-Foundation, 1991).
Overall, the loss of KL expression impairs the resistance of the cells to persistent airway insults and exacerbates irreversible inflammation, which may accelerate the progression of COPD [ 37].
And in a third mechanism, an inappropriate inflammatory response by the innate immune system to transient or persistent non-infectious insults could also trigger a self-perpetuating innate immune response.
Some 27% said they were bullied online, where many were exposed to insults and persistent gossip, and 6% even reported having their personal pictures distorted.
The observation that increased days of febrile illness is correlated with lower developmental scores suggests that persistent or repeated inflammatory insults may have a cumulative effect on developmental outcomes [ 31].
Cofilin actin rods are thought to be an initial protective response to oxidative insults; however, persistent accumulation of rods physically blocks neuritic transport of cargo and impairs synaptic activity.
Accordingly, investigators have focused their attention increasingly on the possible role of another form of myocardial cell death, programmed cell death or apoptosis, as a contributor to overall cardiomyocyte cell death early after ischemic insults following persistent coronary occlusion in patients treated with interventions designed to induce recanalization as promptly as possible.
In these constitutive Atg5 deficient mice, compensatory increase in hepatocytes proliferation and in basal GSH levels as well as faster recovery of GSH content after APAP insult mediated by persistent activation of Nrf2 could account for this apparent discrepancy.
