Sentence examples for persistence of migration from inspiring English sources

An omitted variable bias could hence arise from failing to control for the persistence of migration.

This 2-fold increase in straightness was accompanied by a significant increase in overall displacement or persistence of migration by CT26 cells co-cultured with RAW 264.7 cells.

One source for the differences could have been the fact that most current assays measure a global chemotaxis index that depends on multiple aspects of neutrophil migration, including the fraction of the neutrophil population that are able to move, the alterations of speed, directionality or persistence of migration towards chemoattractant gradients.

Although wild-type and fgf8a −/− cells had similar velocities, we found that fgf8a −/− cells had less-directed migration paths, as measured by both the persistence of migration and the distribution of the angles of migration (Fig. 5K-M).

We believed that the persistence time of migration of fibroblasts would differ depending on their spatial location on the wide fibronectin strip due to the different degree of confinement experienced.

We defined the persistence length of centroid motion as a characteristic length of persistence of directional migration (see Methods).The persistence of directional migration of WT STA cells gradually decays as a function of distance, while that of both pten− STA and pi3k1/2− STA cells, and also of WT VEG cells, rapidly reaches to 0 (Figs.

This breaking of the symmetry of protein localization maintained the leading edge and considerably enhanced the persistence of directed migration, and overall random exploration was ensured by switching among the different ordered patterns.

Moreover, the physical aspects of the ECM such as ECM alignment can alter the persistence of cell migration.

Our analyses indicate that NAV3 decorates the freshly polymerized plus ends of MTs and enhances directional persistence of cell migration.

Myo18a knockdown increased circumferential non-muscle myosin 2A-associated actin filament arrays in the lamella and reduced directional persistence of 2D migration.

Cell-autonomous deletion of GSK-3 reproduced the migration defect and revealed a neuronal morphology defect (the authors argue for dendrite orientation defects), as well as the persistence of the migration defects in postnatal animals.