Sentence examples for persistence of inflammation from inspiring English sources

At 48 h, IL-6/IL-10 ratincreasedsed compared to that at 24 h after CLP, which shows the persistence of inflammation.

At 48 h, IL-6/IL-10 ratincreasedsed compared to 24 h after CLP, what highlights the persistence of inflammation (Fig. 6c).

Still, others report that the persistence of inflammation in IRU is the cause of complications like CME and epiretinal membrane [6, 18].

A final common pathway for many of these insults is initiation and persistence of inflammation in immature lungs [4].

In experimental models of colitis, the appearance of increased numbers of Foxp3+ cells precedes the spontaneous resolution of inflammation, whereas a low number is associated with persistence of inflammation [26].

Since IL-10 is involved in the reduction and resolution of inflammation [25], its lack of production upon T cell contact-activation of monocytes might explain in part the persistence of inflammation in chronic, destructive inflammatory diseases.

Infectious complications and persistence of inflammation-inducing biofilms are, however, other risks of reimplantation of ossicles during cholesteatoma surgery.

Persistence of inflammation-related markers, defined by high circulatory levels of interleukin 6 and 10 (IL-6/IL-10), is associated with a higher post-event mortality rate for CAP patients.

Altogether, these studies suggest that involvement of ILC2s in allergic airway inflammation is unlikely to be limited to the innate immune response but extends to regulation of chronicity and persistence of airway inflammation induced by allergens.

The cellular fraction in the tracheal aspirates is dominated by alveolar macrophages which contribute to the persistence of the inflammation by the production of further pro-inflammatory cytokines and by neutrophils which secrete a plenty of proteases, leading to pulmonary tissue damage, cellular apoptosis, and surfactant inactivation.

These data demonstrate that in a model of allergic lung disease driven by chronic exposure to a clinically relevant aeroallergen, IL-13 plays a significant role in the generation and persistence of airway inflammation, remodeling and dysfunction.