The effects of prenatal stress persist in the absence of stress in the filial generations, suggesting physiological and behavioural programming in the offspring with possibly lifelong consequences.
Nevertheless, despite these favorable developments, significant stresses persist in many markets.
Changes in epigenetic regulation of gene expression induced by environmental exposure can persist in the next sexual generation in stressed animals and plants [1], [2], [3], [4].
In order to combat oxidative and nitrosative stresses and persist in macrophages successfully, M. tuberculosis is endowed with a very efficient antioxidant complex.
Lower levels of the PON1 enzyme, which is involved in protection against OPs and oxidative stress, persist in young children past 2 years of age through at least 7 years of age.
It puts the brakes on cell growth and allows bacteria to persist in the face of starvation, stress, and antibiotics.
By contrast, the limitation on respiration and OXPHOS that is conferred by decreased ANT activity in sesB should persist in the adult, imposing chronic stress and probably accounting for the drastically curtailed lifespan.
Despite the presence of enzymatic and nonenzymatic antioxidant defense mechanisms, oxidative stress may persist in cells as a result of endogenously and exogenously generated ROS, which may lead to damage to macromolecules including DNA, proteins and lipids.
This limits the capacity of the system to persist in its current state in the face of stresses and shocks.
While meeting with authorities in the country, former British Prime Minister Tony Blair stressed that the world needs to persist in its fight against Ebola.
How could these reverted strains persist in our experiment in the presence of the two stresses?
