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De Vlieghere, E. et al. Tumor-environment biomimetics delay peritoneal metastasis formation by deceiving and redirecting disseminated cancer cells.
Hua, K. T. et al. The H3K9 methyltransferase G9a is a marker of aggressive ovarian cancer that promotes peritoneal metastasis.
Peritoneal metastasis is a major cause of morbidity and mortality in ovarian cancer.
de la Fuente, A. et al. M-Trap: exosome-based capture of tumor cells as a new technology in peritoneal metastasis.
Malek JA, Mery E, Mahmoud YA, Al-Azwani EK, Roger L, Huang R, et al. Copy number variation analysis of matched ovarian primary tumors and peritoneal metastasis.
Our experiments suggest the use of a biomimetic trap based on tumor environment interactions to delay peritoneal metastasis.
We aimed to develop a biologically targeted nanoparticle therapeutic for the treatment of ovarian cancer peritoneal metastasis.
Peritoneal metastasis is a major cause of death and preclinical models are urgently needed to enhance therapeutic progress.
Our results suggest that folate-targeted nanoparticles containing chemoradiotherapy have the potential as a treatment for ovarian peritoneal metastasis.
Peritoneal metastasis was not observed.
Fig. 5 Change in peritoneal metastasis near the hepatic flexure.
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