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We also show that peripheral presentation of this same selecting ligand is affected by the activation state of the APCs.
For each peripheral presentation of faces, emotional expression was separated first in the ipsilateral amygdala (Figure 6).
During the whole recording run, subjects fixated the center of the images for central presentation or a fixation cross at the screen center for peripheral presentation.
The frontal activation related to peripheral presentation of fearful faces before 130 ms could be driven by subcortical structures directly connected to the frontal lobe [48].
Structures exhibiting a statistically differential response to stimuli between fearful and neutral faces are listed in Table 1 and Table 2, respectively for central and peripheral presentation condition.
In line with the foveal results described in experiment 1, during peripheral presentation, rivalry dominance was again reported to have very strong and consistent biases at each location.
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The early amygdala response that we observed around 100 ms could result from this subcortical pathway activation for peripheral presentations.
To date, only a limited number of neuroimaging studies have been systematically conducted using peripheral presentations to study the laterality effect on the processing of facial emotions.
We further show two novel and potentially important asymmetries in visual processing: the earlier amygdale activation on the ipsilateral side for peripheral presentations, and the earlier MPFC activation for upper visual field presentation as compared to central and lower visual field presentations.
Caspr2 autoantibodies have especially been associated with peripheral presentations and peripheral motor excitability.
Whereas whole trials included a trial type instruction followed by peripheral stimulus presentation and subject response, half trials included only the instruction component, allowing us to measure instruction-related activation separately from response-evoked signals for both saccades and nogo trials.
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