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Peripheral mechanisms also play a major role in TTH.
Originally, two theories involving peripheral mechanisms were proposed.
Our results support peripheral mechanisms are maintained in multifocal NH.
This includes both central and peripheral mechanisms, as well as nerves and vessels.
The pathomechanism of CH is not fully understood but involves both central and peripheral mechanisms [2].
Peripheral mechanisms are essential causes of thrombocytopenia [15, 214, 218, 227, 232, 233].
Currently, there is no other test available to distinguish between central and peripheral mechanisms.
The loops interconnect central and peripheral mechanisms and coordinate their functions.
It remains unclear if TTH is caused by peripheral mechanisms or by the central nervous system (CNS) [5, 6].
However, it is becoming increasingly evident that NTG exerts its hyperalgesic effect through central and peripheral mechanisms [15, 16].
However, thrombocytopenia in the ICU setting is often multifactorial [9], frequently involving both central and peripheral mechanisms.
Related(19)
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remote mechanisms
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adjacent mechanisms
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secondary mechanisms
accessory mechanisms
peripheral organizations
isolated mechanisms
far mechanisms
ancillary mechanisms
peripheral arrangement
peripheral system
peripheral flaws
peripheral ways
peripheral pulses
peripheral resentments
peripheral neuropathies
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