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Furthermore, an early increase of perfusion in the base of both lungs was observed and sustained after BMDMC administration.
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An early increase in perfusion at the base of both lungs was observed and sustained after BMDMC administration.
Even though no significant changes were reported in lung function and quality of life, autologous BMDMC transplantation seems to have led to an early increase in perfusion at the base of both lungs, which remained increased for the duration of the follow-up.
Media perfusion from the SUB to the base of the chamber (typically 380 mL/min, Watson Marlow peristaltic pump) allowed fluidisation of the beads (1.675-fold 1.675-fold).
Type I was lack of central blood perfusion with sufficient peripheral blood perfusion; Type II was heterogeneity blood perfusion and much less blood perfusion in the area of necrosis; Type III was lack of central and peripheral blood perfusion.
An example of the key perfusion time steps – RV enhancement, LV enhancement, and myocardial perfusion – at the base level is given in Figure 1.
We present an anatomically based theoretical model of perfusion in the acinar blood vessels designed to investigate changes in PVR following occlusion of arteries <500 μm in diameter.
The two peaks of perfusion in the normal hindlimb appeared to correspond to macrovasculature regions with high perfusion rates and microvascular compartments with lower perfusion rates.
At perfusion rates up to 130 mL min−1, the extent of endothelial cell coverage, calculated from CD31 + endothelial cell linear coverage post perfusion/CD31 + endothelial cell linear coverage pre perfusion, in the human umbilical vein remained intact during 6 h of perfusion (Fig. 4b).
First, the estimated tumor perfusion in ASL was based on assuming a constant value of tissue/water partition coefficient, λ.
The location of the perfusion center of gravity will depend on the relative perfusion in the left ventricle.
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