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Perfusion block.
Perfusion block did not damage healthy lungs.
Perfusion block of these preconditioned lungs caused considerable pathological changes that were more pronounced following the lower lobe perfusion block on the left.
Somewhat unexpectedly, the lower lobe perfusion block alone did not cause any physiological or radiological abnormalities.
Three animals with protective ventilation and perfusion block of the right lower lobe.
Perfusion block of the left lower lobe was much more detrimental to hemodynamics, lung mechanics, tissue aeration, and fluid content in the contralateral lung than the corresponding effects caused by perfusion block of the right lower lobe.
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The right and left perfusion blocks caused the same "intensity" of edema in conditioned lungs.
In vitro, 300 µM DL-TBOA given 10 minutes prior to and during 10 mM [K+] perfusion completely blocked glutamate transport reversal-induced D-[3H]aspartate release, verifying that DL-TBOA is an effective inhibitor of glutamate transport reversal (Fig. 7c).
The anti-inflammatory effects of URB597 on leukocyte rolling and vascular perfusion were blocked by both CB1 and CB2 antagonism, while the effect on leukocyte adherence was independent of cannabinoid receptor activation.
Upon completion of the last SR block, perfusion imaging was started again with duration of 16 min (Post).
The embolus will block perfusion to lung units, converting them into VDalv [ 1, 25].
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