This observation may indicate that the auditory and visuospatial tasks are actually too similar to draw conclusions about general effects on working memory per se.
Because most of the available evidence is derived from tasks not specifically developed to probe working memory per se, we wanted to investigate whether working memory deficits may be detected across different paradigms [ 1].
The results have implications for the cognitive impairments seen in illnesses characterized by chronic cytokine activation (e.g., Alzheimer's disease) as well as illnesses treated with cytokines (e.g., multiple sclerosis) suggesting that some cognitive failures attributed to working memory impairments per se may better be attributed to prior attention impairments.
First, the possibility remains open that it is not working memory involvement per se that is the critical factor, but an executive function that might also be involved in accessing or updating working memory.
Over the last 20 trials of training they averaged approximately one working memory error per trial (mean – 1.01 ± 0.32).
Thus our detection task was designed to be as simple as possible so that errors would be unlikely to be attributable to deficits in visual processing, perception, working memory or neglect per se.
Also in pre-school children, visuospatial working memory training improved untrained verbal working memory performance.
The results show that most of the activity typically elicited by no-go signals, including pre-SMA hemodynamic response, is actually driven by the engagement of high attentional or working memory resources, not by inhibitory processes per se.
At the end of 2002, a neurologist treated him with 50 mg methylphenidate per day for suspected ADHD, a diagnosis related to difficulties with working memory and attentional tasks identified in a neuropsychological assessment.
We evaluated two strategies for alleviating working memory load for users of voice interfaces: presenting fewer options per turn and providing confirmations.
