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Conclusion PEEP decrease during prone position as a consequence of improvement of oxygenation has a marginal but significant positive effect on cardiac index, related to an increase in global end-diastolic volume and probably venous return.
The expiratory and inspiratory resistance and the intrinsic PEEP decrease significantly after fenoterol administration by MDI in ventilated COPD patients.
Completion of volume change in 99 % of the patient's PEEP decrease events required 29 breaths (1 min, 56 s) (Fig. 3).
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Although high IAP-matching PEEP decreased cardiac output, moderate IAP-matching PEEP provided a compromise between lung function and hemodynamics.
In animals, PEEP decreases hepatosplanchnic perfusion in a dose-dependent manner, with a limited effect at PEEP levels of less than 10 cmH2O [ 2, 4, 5].
High PEEP decreases splanchnic perfusion [1, 5, 20].
PEEP decreases cardiac output by limiting right ventricular filling and increasing right heart afterload.
PEEP decreased by at least 5 cm of H2O in 18 (12%) of the PP sessions.
The incremental application of 5 to 15 cmH2O PEEP decreased cardiac output but improved respiratory system compliance and returned venous admixture to pre-injury levels.
Five cmH2O PEEP decreased the spectral power increment to approximately half of its injury-induced value, while 10 cmH2O PEEP abolished the effect of injury on the acoustic power spectra.
Increasing PEEP decreased the C20/C ratio and increased %E2.
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