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Posterior reversible encephalopathy syndrome (PRES) relates to a transient pattern of brain edema mostly involving the parietal and occipital regions.
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Echographic patterns suggestive of brain edema and/or periventricular hyperechogenicity were observed in 28 out of 48 control infants with perinatal asphyxia and in 10 out of 12 infants of the neonatal Ominous Outcome Group.
Evaluation of brain edema caused by influenza associated encephalopathy (IAE) model using Evans blue administration.
These results indicate that the mechanism of brain edema induction regulated by Setdb2 might be different between LPS group and IAE group.
Nosaka, N. et al. Anti-high mobility group box-1 monoclonal antibody treatment of brain edema induced by influenza infection and lipopolysaccharide.
To evaluate whether IAE was induced or not, we examined the occurrence of brain edema by intravenously administering Evans blue dye 2 h before euthanization.
The pathogenesis of brain edema in patients with chronic liver disease (CLD) and minimal hepatic encephalopathy (HE) remains undefined.
Cerebral water accumulation was studied during induction of brain edema in dystrophin-null transgenic mice (mdx-betageo) and control mice.
It showed two peaks of brain edema formed after TBI.
To reveal the degree of brain edema, brain water content was detected.
Inhibition of brain edema therefore plays a vital role in the management of malignant gliomas.
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