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The different distribution pattern might be explained both by different MB composition and the attached ligands.
This species specific reaction pattern might be explained by the isohydric character (McDowell et al. 2008; Hartmann 2011) of Norway spruce.
This pattern might be explained by the model of Ormel et al. [ 82].
Differences in the number, clustering, and distribution pattern might be explained by distinct strategies of neurogenesis among respective taxa.
This pattern might be explained by the higher amplification rate of Ty1- copia (compared to Ty3- gypsy) in recently diverged species.
This different pattern might be explained by items being somewhat disparate, covering questions about voice quality as well as personality, and therefore of differing relevance.
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Co-option of a new signaling pathway (Wnt) in mesoderm patterning might be explained by a need for increased complexity and quantity of ventral mesoderm in vertebrate development.
Although, loss of variation of the Y chromosome in the bonobo by a founder effect or genetic drift cannot be excluded, these contrasting patterns might be explained in the context of the species' markedly different social and mating behaviour.
These patterns might be explained by high selectivity of AMF by their hosts.
These complex patterns might be explained by the existence of higher order interactions involving more than two loci or simply because small sample size for certain genotypic classes might lead to inaccurate estimates of the epistatic effects.
Although the single process of bone remodeling might explain these different patterns, discrepancies might be explained by the fact that remodeling would occur in different contexts.
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