Sentence examples for pathways to block from inspiring English sources

Our results demonstrated that AcNOS may be another effector gene in addition to prophenoloxidase pathways to block the development of the malaria parasite in An. culicifacies mosquito's midgut lumen and thus may elucidate a novel putative mechanism of refractoriness.

Oestrogen may act through one of these pathways to block SOX9 nuclear entry in marsupial gonads.

Rheumatoid arthritis synovial cells are just one example where TNF appears to use such pathways to block Fas-mediated programmed cell death [ 58, 59].

All in all, in vitro studies showed the possible promising functions of atorvastatin, simvastatin, and pravastatin as the representatives of the statin family, on several cellular pathways to block the virus-host interaction system, which can act as an effective compound opposed to H1N1 infection.

It is thus inviting to speculate that NGF plus Col 1 and UG26 CM may modulate similar downstream pathways to block apoptosis, as suggested by our finding of a differential expression of genes in the "Apoptosis" REACTOME pathway following UG26 CM exposure, and the similar relative decrease in Annexin V staining of ESLAM cells incubated with either of these additives.

This suggests that Plk1 is acting early in the contractile ring assembly pathway to block both anillin and myosin II localization and thus the primary effect of Plk1 inhibition is likely on the Rho pathway.

In prostate cancer as well as in numerous types of cancer, attempts at defining novel or alternative "druggable" pathway to block tumor progression converged toward growth factors signaling pathways, for example, invasion, lymphangiogenesis, and angiogenesis [ 21, 22].

The Inhibitor of Apoptosis proteins (IAP) are a family of novel genes that function in the cell death pathway to block apoptosis induced by a variety of triggers [reviewed in [ 2- 4]].

Tobacco cells expressing a prenylatable GFP were treated with specific MEP and/or MVA pathways inhibitors to block the formation of prenyl diphosphates and therefore the possibility to modify the proteins.

Inhibition of both pathways synergised to block the growth of the cells.

However, inhibitors of the ERK and JNK pathways failed to block the effect of cis-UCA on cell viability.