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The insights from these pathways can provide valuable input for Jiangsu's future energy policies.
Perturbing plants by mis-regulating key genes/enzymes integral to major cell wall pathways can provide both rich insights into cell wall development and architecture, and concurrently provide significant opportunities for improved lignocellulose utilization.
Elucidation of the relevant biosynthetic pathways can provide increased access (e.g., via molecular breeding or metabolic engineering) and enable reverse genetic approaches toward understanding the physiological role of these natural products in plants as well.
Modern detailed chemical mechanisms containing detailed descriptions of the elementary reaction pathways can provide predictive modeling of fuel chemistry; however, they are generally far too computationally expensive for use in CFD and computational engine design and are often only available for single fuel components or simple component mixtures.
Precise knowledge of optimal gene regulatory pathways can provide an understanding of the time-dependent enhancement and suppression of gene activity and drug effectiveness [20] [22].
Moreover, the information of up- and down-regulation of those genes as well as their positions in the pathways can provide indication of the positive or negative outcome of the pathway [34], [35], which is instrumental to estimate the impact exerted by the differentially expressed genes on the pathway.
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Simultaneous identification of multiple metabolites, and information about their coexistence within a single pathway can provide clues about the relationship between development or environmental interactions, and the metabolic adjustments that accompany such events.
This pathway can provide SAM, the precursor molecule needed for nicotianamine biosynthesis.
The glycolysis pathway can provide carbon skeletons to the TCA cycle, lipid metabolism and phenylpropanoid-flavonoid pathway.
Feeding experiments have confirmed that the mev pathway can provide some or all of the isoprene incorporated into HI secondary metabolites [ 23, 27– 30] and led to the hypothesis that the mev pathway is a marker for HI production [ 26].
Also, genetic epidemiological studies of biological phenotypes involved in the same pathway can provide relevant information, and can contribute to unravel the mechanisms underlying complex diseases such as OSA.
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