Sentence examples for pathway dna from inspiring English sources

The methyldiazonium cation creates DNA adducts a marker of TMZ PD that trigger DNA damage responses involving the p53 pathway, DNA repair mechanisms, and potentially cell-cycle arrest and cell apoptosis.

The Central Dogma of molecular biology is that pathway: DNA -> messenger RNA -> protein.

Cellular effects of RF-EMF exposure reportedly involve the apoptotic pathway, extracellular signal pathway, DNA damage response, cell proliferation, and cell cycle3,12,13,14,15.

These genes function in different facets of the p53 signalling pathway; DNA repair and damage (Sesn1, Sesn2, Gadd45, Igfbp3, Ampkb), suppression of reactive oxygen species and protection from oxidative stress (Sesn1, Sesn2), IGF (Igfbp3), mTOR (mammalian target of rapamycin) and autophagy pathway (Sesn1, Sesn2, Ampkb).

These pathways mainly include p53 pathway, Wnt pathway, HIF pathway, DNA replication and repair pathway, cyclins and cell cycle regulation, and oxidative stress pathways.

A variety of signal pathways are activated in different types of cancer cells, such as p53 pathway, Wnt pathway, HIF pathway, DNA replication and repair pathway, cyclins and cell cycle regulation, EGF pathway, AKT pathway and VEGF pathway.

Another intriguing possibility was recently put forward by Javid et al. [ 70], who argue that protein variability driven by errors in the central information-processing pathway (DNA-RNA-protein) may provide a phenotypic stepping-stone to resistance akin perhaps to other ancillary mechanisms shown in Fig.  1.

In this review we try to concentrate on very specific pathways (DNA damage response, DDR, and epigenetic modifiers) and very specific determinants (senescence-associated secretory phenotype, SASP-miRNAs) of human premature aging.

Depending on the type of cell and the status of cell cycle checkpoint pathways, DNA damage produced by replication stress could induce a number of aging phenotypes.

These genes were involved in one or more critical organism process such as apoptotic pathways, DNA repair, development or inflammation.

We describe the changes in expression of genes relating to primary metabolic pathways, DNA synthesis, late embryogenesis proteins, and embryo storage proteins.