Sentence examples for pathology table from inspiring English sources

Subsequently other authors have reported cases of RES secondary to an underlying pathology (Table 1) [7, 9, 10, 16].

Eight reports of gastric lipomatosis exist in the literature, of which five describe a detailed pathology (Table 1) [2, 5, 6, 8 12].

Thus, cells from embryo/fetus of Atg7−/− mice and certain cell/tissue-specific Atg7-knockout postnatal mice were extensively employed in the quest for understanding the mechanisms underlying the pleiotropic effects of ATG7 in development, physiology, and pathology (Table 1).

The majority of patients had additional low-grade (Gleason 3+3) cancers at final pathology (Table 1), but such low-grade disease was not the subject of this study.

These percentages were calculated for each pathology (Table 4), showing that some pathologies had a high rate of exclusion (e.g., pneumonia/TB, splenomegaly, uterine myomatosis, ovarian cyst, cirrhosis, gastritis/PUD).

Obviously, changes were observed in clinical chemistry features, including ALP, TP, and TC, and in liver pathology (Table 1 and Figure 2, respectively), suggesting that SWCNTs clearly have hepatotoxic abilities in rats.

VOIA50, VOIRTL and GradWT2 did not show any outliers, provided only small, non-significant differences in maximum tumour diameter (<4.7 mm, p>0.10), and showed fair correlation (R2>0.62, slope 0.88-0.97) with pathology (Tables 1 and 2).

In the 10-week experiment (Table 1), bacterial colony forming units per gram (CFU/g) of liver and spleen was greatest at three weeks post-infection, consistent with the time-point of most severe HLH pathology (Tables 1 2), and decreased by six and 10 weeks post-infection.

> > -wrap-foot> Mice heterozygous for semi-dominant Col4a1 or Col4a2 mutations have multi-system disorders including ocular, renal, pulmonary, muscular, vascular, reproductive and central nervous pathology (Tables  4 and 5).

To test this hypothesis, we analysed expression of hypoxia-related genes in the central and peripheral retina from 13 human donors between the age of 17 and 92 years without diagnosed retinal pathologies (Table S1).

Their multivalent roles in delaying cellular senescence and blocking the development of premature aging phenotypes further mark them as promising targets to design intervention for several age-associated pathologies (Table 1).