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These latter findings suggest that striatal pathology may be a primary driver of these secondary pathological events.
An alternative reliable tool in the differential diagnosis of pulmonary pathology may be bedside lung ultrasonography.
Peripherally located frontal sinus pathology may be unreachable with standard endoscopic techniques.
Pathology may be highlighted but the anatomic data captured by volumetric imaging is not altered (Fig. 2).
Gurvits et al. reported that coronary artery disease, recent surgery, and gastroduodenal pathology may be associated with esophageal stricture [1].
Systemic pathology may be caused either through direct virus-mediated infection or by indirect mechanisms such as 'by-stander' apoptosis.
The extent of the liver pathology may be related to other patient specific factors such as obesity, diabetes and presence of metabolic syndrome [2].
Low-grade inflammation and microvessel pathology may be responsible for initiating or exacerbating some of the hearing loss associated with aging.
Although the pathology may be asymptomatic and discovered incidentally on routine imaging, compression of the cord and surrounding nerve roots can produce myelopathic or radiculopathic symptoms that are best treated with surgical decompression.
Addition of biologics (growth factors and cells) may improve future outcomes, and ultimately a tissue-engineered solution is anticipated, but in the end, host articular cartilage pathology may be a significant confounder of outcome.
This peripheral pathology may be responsible for some of the phenotypes exhibited by the two mouse models.
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