Associations between diabetes and clinical AD could reflect additive effects of subclinical vascular disease (elevated by diabetes) and AD pathology (that is perhaps independent of diabetes).
However, therapies to influence TGF-β2 activity have resulted in clinical benefits to patients with pathologies in which this isoform is perhaps more dominant.
Composing a scholarly treatise regarding the disappearance of pathologies that were prevalent formerly is perhaps more challenging than describing the panoply of CNS changes observed before the era of cART.
This is perhaps not surprising as it has been shown that responses to ESAT-6 correlated positively with the degree of pathology [30].
However, the magnitude of the change required for a beneficial effect on pathology is unknown and perhaps the levels of rescue achieved meet a therapeutic threshold.
But the field of coral pathology is in its infancy, with perhaps only five of what could be dozens of diseases linked to specific microbes so far.
The damage is likely to continue to mount, perhaps because the underlying pathology is hardly restricted to those who have taken the fall.
Perhaps the most striking feature of the prion-induced synaptic pathology is the increased curvature of the PSD.
Pathology The pathology is not known.
Even his pathology is ordinary.
