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A rising awareness of the complexity of epileptic pathology is, however, brought about by the emergence of nonreductionist systems biology (SB) that considers the networks of interactions underlying the normal organismic functions and of SB-based systems (network) pharmacology that aims to restore pathological networks.
The relationship between clinical expression of the disease and progression of the microvessel pathology is, however, uncertain as we lack tools for imaging brain vessels in vivo.
The distribution of alpha-synuclein pathology is however different in M83-, M20-, and wild-type mice that have been injected with recombinant alpha-synuclein fibrils and in these cases can more strongly involve the cortex [ 28, 48].
This comprehensive approach to joint pathology is, however, far from the norm across different species and models, and methods for quantifying histopathology in joint tissues other than cartilage have not been standardized [ 87].
The relevance of the relatively mild increase in the frequency of point mutations we observed for these variants in the yeast model for the human pathology is, however, not evident.
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Eating disorder pathology was, however, strongly associated with depression and low self-esteem (7).
How one should treat coexisting acetabular labral pathology is still the subject of debate, however.
However, data on the frequency, impact, and predisposing factors for this pathology are still rare.
The tau pathology we detect is, however, less robust compared to JNPL3 mice in a mixed background [ 36].
The significance of the lack of P986 3-hydroxylation in the pathology is uncertain, however.
With the focus of this vaccine on the RBD of MERS, the possibility of vaccine-induced immune pathology is reduced; however, in vivo testing is required to confirm this result.
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