Sentence examples for pathology control from inspiring English sources

Patients were divided into four groups, according to the intraoperative findings, as follows: Group I: no shoulder pathology (control group); Group II: partial thickness rotator cuff tear; Group III: full thickness rotator cuff tear; Group IV: cuff arthropathy.

In parallel with the described modifications of the blood vascular system, the reactive LN can also undergo substantial changes in the local lymphatic apparatus and lymphatic drainage, processes that are now the object of growing attention in the field of chronic inflammatory diseases as potential check-points of peripheral pathology control (see Section 4).

The data supported previous findings from a study investigating neuropathologic and biochemical differences in the oldest old, which evaluated nondemented pathological aging (referred to as high pathology controls) [ 3].

While increasingly recognized, there is a range of terms for pathological aging that makes review of the literature challenging, including presymptomatic or incipient AD [ 4], preclinical AD [ 6], pathological AD [ 7], nondemented high pathology controls [ 3], asymptomatic persons with AD neuropathology [ 10], intermediate probability mismatches [ 11], and pathologically preclinical AD [ 5].

Post-mortem analysis in human subjects disclosed that Aβ(1 40) rather than Aβ(1 42), whether in soluble or in insoluble form, discriminated more readily between AD patients and high pathology controls [8].

This engagement of the complement system has not been observed in the brains of high pathology controls, contributing to evidence of the involvement of acute mediators in AD.

Fifth, high pathology controls (individuals who have Aβ and tau aggregates at levels similar to AD patients, but do not develop dementia) show lower signs of inflammation [ 41].

The latter group may parallel the group we have called asymptomatic AD at autopsy (and others have called high pathology controls or preclinical AD), because they do not show accelerated cognitive decline despite substantial amyloid pathology [ 11].

We were able to apply DiI-tracing in two human autopsy cases with mild AD-related pathology (controls), in one AD-patient, in one non-demented patient with advanced AD-related pathology, and in three cognitively impaired AGD-patients.

We then used these primer pairs for transcript-specific real-time PCR amplification of RNA harvested from the temporal lobe of 190 human brains without gross pathology (controls) obtained from the Harvard Brain Bank.

DiI application into the entorhinal cortex of the two non-demented patients with low levels of AD-related pathology (controls) marked a high number of neurites projecting into area 35 of the temporal cortex as well as 12 18 retrogradely labeled pyramidal neurons in layers III and V in a 1-mm-thick slab of the area 35 (Figs.  2, 3; Table  1).