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Exact(4)
MIC values of the tested compounds 1, 3, 4, 7, 8 and 10 against pathogens, were determined by microdilution method [40].
IgG, IgA, and IgM antibodies to C. pneumoniae and IgG antibodies to other pathogens were determined using commercially available enzyme-linked immunosorbent assay (ELISA) kits (5).
The susceptibilities of the isolated pathogens were determined by the modified Kirby-Bauer disc diffusion method with Muller Hinton agar plates [ 37].
Of the 234 (79%) outbreaks for which a pathogen was identified, 174 (59%) pathogens were determined to be ENT/HRV (representing 531 positive samples) and were temporally spread throughout the surveillance period.
Similar(56)
The minimum inhibitory concentration (MIC) and minimum bactericidal concentration (MBC) of EEO against the four pathogens was determined by the two-fold dilution method described by Kubo et al. ([2004]), with slight modification.
More specifically the presence of oral marker bacteria and VAP associated pathogens was determined by PCR and the usefulness of DGGE community profiling for the analysis of the endotracheal tube biofilm was assessed.
Whether integrin α3β1 plays a similar role in facilitating responses to live bacterial pathogens was determined by testing responses to B. burgdorferi, a bacterium characterized by its high concentration of lipoproteins.
Host specificity of microbial pathogens is determined by elaborate molecular interactions between the pathogens and hosts.
Presence of the pathogens was determined by PCR analysis and sequence alignments with Genbank (see Methods).
It is now clear that the host specificity of bacterial pathogens is determined by multiple molecular interactions between the pathogens and their hosts.
The prevalence of IgG and IgA antibodies against these pathogens was determined in all sera except against B. afzelii where only the prevalence of IgG was determined.
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