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The emergence of diseases resulting from pathogenic parasites is always an issue of concern in tropical regions.
With regard to oyster infection, Fisher and Newell (1986) pointed out that high salinity reduces the oysters' defense capacity and leaves them more susceptible to pathogenic parasites.
The result of this study clearly demonstrated that the high temperature produced in the thermophilic phase of the composting process is much more effective in sanitizing pathogenic parasites of faecal sludge than the earthworms did.
More than half of the global human population is exposed to the risk of infection spread by mosquitoes; including Culex spp., Anopheles spp. and Aedes spp. that are considered a public health problem, sin are vectors of pathogenic parasites.
Take for example the glycolytic enzyme triosephosphate isomerase (TIM) from two evolutionarily related pathogenic parasites, Trypanosoma brucei and T. cruzi.
The recent decoding of the genome sequences of the two most pathogenic parasites, S. mansoni and S. japonicum, has paved a pivotal way for a systematic dissection of the parasite biology [2], [3], [4].
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This enzyme, recently proven to be essential for this pathogenic parasite, shares 33% sequence identity with the corresponding enzyme in the human host.
Chagas named the pathogenic parasite as Trypanosoma cruzi and later that year as Schizotrypanum cruzi, both honoring Oswaldo Cruz, the noted Brazilian physician and epidemiologist who successfully fought epidemics of yellow fever, smallpox, and bubonic plague in Rio de Janeiro and other cities in the beginning of the 20th century.
The discovery of mating for this highly prevalent and pathogenic parasite of livestock has complex but considerable consequences for the spread of traits such as drug resistance.
In the present study we investigated the effects of the potent, third generation P-gp inhibitor GF120918 in the pathogenic parasite T. gondii.
This phenotype characterizes the individual tendency to become clinically ill following P. falciparum infection; 2) The maximum and mean P. falciparum trophozoite parasite densities (i.e. pathogenic parasite stage infecting red blood cells) during clinical episodes (acronym mx-PFD and me-PFD, respectively).
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