Indeed, our understanding of the pathogenesis, clinical spectrum and epidemiology of GORD has continuously evolved.
Available evidence implicates environmental factors in the pathogenesis of autism spectrum disorders (ASD).
Enhanced mammalian target of rapamycin (mTOR) signaling in the brain has been implicated in the pathogenesis of autism spectrum disorder (ASD).
These findings may have important implications for OL biology and for our understanding of the pathogenesis of a spectrum of dysmyelinating and demyelinating diseases as well as processes that may be recapitulated during the process of remyelination following injury or disease states [65], [66].
Despite the 20 years that have elapsed, the clinical spectrum, pathogenesis, and possible treatment for ICL remain obscure.
This gene is significant in innate immunity and the LTB4/LTB4R pathway is involved in the pathogenesis of the spectrum of human inflammatory diseases.
This observation might be important in the pathogenesis of autism spectrum disorder (ASD) in which the genetic as well as important environmental effects might be involved.
Despite advances in our knowledge of the pathogenesis of leprosy spectrum, the understanding of the mechanisms of nerve damage and regeneration in leprosy-associated neuropathy remains poor.
Even though it is not possible to completely rule out the effect of secular environmental changes related to the pathogenesis of autism spectrum disorder, the results indicate that their effect over the past decade may be marginal.
To analyze the clinical spectra, pathogenesis, treatment, outcome, and prognostic factors of fungal empyema thoracis.
