The phrase "pathogenesis of diseases" is correct and usable in written English.
It can be used in medical or scientific contexts when discussing the development and progression of diseases.
Example: "Understanding the pathogenesis of diseases is crucial for developing effective treatments and preventive measures."
Alternatives: "development of diseases" or "origins of diseases."
Apoptosis and necrosis have been extensively studied during the development of organisms and the pathogenesis of diseases.
As such, osteoclasts have been implicated in musculoskeletal tissue damage and the pathogenesis of diseases characterized by inflammatory osteolyis, including RA, psoriatic arthritis, and peridontitis.
Background: The role of oxidative stress in the pathogenesis of diseases such as macular degeneration, certain types of cancer, and Alzheimer's disease has received much attention.
Oxidative stress has been thought to play an important role in the pathogenesis of diseases such as cancer, cardiovascular disease, atherosclerosis, diabetes mellitus, and neurodegenerative disorders (Valko et al. [2007]).
Some of these sequences constitute the pathogenesis of diseases like AD. Microglia are the critical convergence point for the many diverse triggers in orchestrating the activity of other immune cells in the brain [6].
Edible mushrooms had been used in the treatment of different ailments in folk medicine and possesses secondary metabolites that could treat or slow down the pathogenesis of diseases like liver and kidney pathologies [3, 4].
Recent systematic gene inactivation studies have confirmed the integral role of two-component systems in the pathogenesis of diseases caused by several microorganisms and highlighted the validity of using these systems as targets for therapeutic intervention.
Such an approach is possible thanks to the knowledge on: pathogenesis of diseases at molecular level, metabolism of healthy and affected cells as well as metabolism of microorganisms (bacteria, fungi, protozoa, etc).
Likewise, the pathogenesis of diseases involving hyperuricemia likely involve uric acid's direct and indirect pro-oxidant properties.
The signaling pathways that may modulate the pathogenesis of diseases induced by expanded polyglutamine proteins are not well understood.
The enzymatic activity of TG2 is of pharmacological interest because it is believed to contribute to the pathogenesis of diseases such as celiac sprue [6], neurodegenerative disorders [7], type 2 diabetes [8], and certain cancers [9] [11].
