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It remains to be investigated whether the observed effects result from direct actions of translocated particles or specific components (e.g. PAH, metals, VOC) carried on the surface of these particles (Donaldson et al. 2005), or whether they are caused indirectly by mediators released from respiratory tract and/or the blood (Peters et al. 2006).
It is also possible that BS gives a better measure than PM10 for the more damaging elements of air pollution, e.g., fine or ultrafine particles or specific transition metals (Heal et al. 2005; Hochadel et al. 2006; Janssen et al. 2011).
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Relating the inflammatory effects to the particle characteristics—organic content, primary particle size, or specific surface area demonstrates the most obvious dose response for particle surface area.
The primary particle size or specific surface area, together with loading, determines the effective contact area between the filler and rubber matrix.
Due to the small diameter of ENM, it is questioned if the approach of using a mass basis is adequate for ENM or if the particle number or specific surface area should to be taken instead.
NPC HONE1 and SUNE1 cells were infected with lentiviral particles containing specific or negative control vectors, and the polyclonal cells with GFP signals were selected for further experiments using FACS flow cytometer assay.
NPC 6-10B cells were infected with lentiviral particles containing specific or negative control vectors, and the polyclonal cells with GFP signals were enriched for further experiments using FACS flow cytometer assay.
NPC 5-8F cells were infected with lentiviral particles containing specific or negative control vectors, and polyclonal cells with GFP signal were selected for further experiments using FACS flow cytometery.
LD, also named lipid particles or oil bodies are specific subcellular compartments which gained much interest recently regarding their emerging role in health and disease [7].
From results generated by multiple groups, it became clear that interference can be caused by either inactivated virus particles or live virus grown under specific conditions, such as repeated passage with large inocula or repeated freeze-thawing.
Studies of viral attachment to cultured gastrointestinal epithelial cells (Caco-2) using recombinant virus-like particles or infectious noroviruses indicate that specific histo blood group antigens play a key role in the attachment of the virus to the host cells (13 – 17 ).
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